[1] WHITEHEAD C C, FLEMING R H. Osteoporosis in cage layers[J]. Poult Sci, 2000, 79(7):1033-1041.
[2] BAIG M, TARIQ S, TARIQ S. Homocysteine and leptin in the pathogenesis of osteoporosis-evidences, conflicts and expectations[M]//Dionyssiotis Y. Advances in Osteoporosis. Rijeka:InTech, 2015.
[3] BLOUIN S, THALER H W, KORNINGER C, et al. Bone matrix quality and plasma homocysteine levels[J]. Bone, 2009, 44(5):959-964.
[4] ENNEMAN A W, SWART K M A, ZILLIKENS M C, et al. The association between plasma homocysteine levels and bone quality and bone mineral density parameters in older persons[J]. Bone, 2014, 63:141-146.
[5] KALANI A, KAMAT P K, VOOR M J, et al. Mitochondrial epigenetics in bone remodeling during hyperhomocysteinemia[J]. Mol Cell Biochem, 2014, 395(1-2):89-98.
[6] VACEK T P, KALANI A, VOOR M J, et al. The role of homocysteine in bone remodeling[J]. Clin Chem Lab Med, 2013, 51(3):579-590.
[7] VIJAYAN V, KHANDELWAL M, MANGLANI K, et al. Homocysteine alters the osteoprotegerin/RANKL system in the osteoblast to promote bone loss:pivotal role of the redox regulator forkhead O1[J]. Free Radic Biol Med, 2013, 61:72-84.
[8] DAYAL S, LENTZ S R. Homocysteine:a controversial cardiovascular risk factor[M]//WANG H, PATTERSON C. Atherosclerosis:Risks, Mechanisms, and Therapies. London:John Wiley & Sons, Inc, 2015:53-62.
[9] ETCHES R J. Calcium logistics in the laying hen[J]. J Nutr, 1987, 117(3):619-628.
[10] MACLEAN K N, KRAUS E, KRAUS J P. The dominant role of Sp1 in regulating the cystathionine β-synthase -1a and -1b promoters facilitates potential tissue-specific regulation by Kruppel-like factors[J]. J Biol Chem, 2004, 279(10):8558-8566.
[11] ZHAO J Y, YANG X Y, SHI K H, et al. A functional variant in the cystathionine β-synthase gene promoter significantly reduces congenital heart disease susceptibility in a Han Chinese population[C]//International Meeting on Human Genome Variation & Complex Genome Analysis. 2012.
[12] 胡慧艳,贾青,侯胜奎,等.猪DKK1基因启动子区的克隆及其活性分析[J].畜牧兽医学报,2017,48(6):1150-1157.
HU H Y, JIA Q, HOU S K, et al. Cloning and activity analysis of the promoter region of swine DKK1 gene[J]. Acta Veterinaria et Zootechnica Sinica, 2017, 48(6):1150-1157.(in Chinese)
[13] SAXONOV S, BERG P, BRUTLAG D L. A genome-wide analysis of CpG dinucleotides in the human genome distinguishes two distinct classes of promoters[J]. Proc Natl Acad Sci U S A, 2006, 103(5):1412-1417.
[14] ATTWOOD J T, YUNG R L, RICHARDSON B C. DNA methylation and the regulation of gene transcription[J]. Cell Mol Life Sci, 2002, 59(2):241-257.
[15] 涂小煌, 黄少雄, 李文生, 等. 胱硫醚-β-合酶基因启动子区CpG岛甲基化水平与结直肠癌患者临床病理特征的关系[J]. 中华肿瘤杂志, 2013, 35(5):351-355.
TU X H, HUANG S X, LI W S, et al. Correlation of methylation of CpG island in cystathionine beta synthase promoter and clinicopathological features in colorectal cancer[J]. Chinese Journal of Oncology, 2013, 35(5):351-355. (in Chinese)
[16] ZHAO H, LI Q S, WANG J, et al. Frequent epigenetic silencing of the folate-metabolising gene cystathionine-beta-synthase in gastrointestinal cancer[J]. PLoS One, 2012, 7(11):e49683.
[17] ZHANG Z Z, HE Y, TU X H, et al. Mapping of DNA hypermethylation and hypomethylation induced by folate deficiency in sporadic colorectal cancer and clinical implication analysis of hypermethylation pattern in CBS promoter[J]. Clin Lab, 2017, 63(4):733-748.
[18] HUSAIN A, JEFFRIES M A. Epigenetics and bone remodeling[J]. Curr Osteoporos Rep, 2017, 15(5):450-458.
[19] DUTTENHOEFER F, BISWAS S K, IGWE J C, et al. Sp1-dependent regulation of PPARα in bone metabolism[J]. Int J Oral Maxillofac Implants, 2014, 29(1):e107-116.
[20] REUMANN M K, STRACHNA O, LUKASHOVA L, et al. Early growth response gene 1 regulates bone properties in mice[J]. Calcif Tissue Int, 2011, 89(1):1-9.
[21] MANN V, RALSTON S H. Meta-analysis of COL1A1 Sp1 polymorphism in relation to bone mineral density and osteoporotic fracture[J]. Bone, 2003, 32(6):711-717.
[22] GRANT S F A, REID D M, BLAKE G, et al. Reduced bone density and osteoporosis associated with a polymorphic Sp1 binding site in the collagen type I α 1 gene[J]. Nat Genet, 1996, 14(2):203-205. |